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ON THIS PAGE:
Tanya's Story
Thomas's
Story
Ollie's Story
HOME
Site Overview
What You Need to Know
First
Alphabetical Index
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WHAT IS
CKD?
What Happens in
CKD?
Causes of CKD
Early Detection
How Bad is It?
Is There Any
Hope?
Acute Renal
Failure
KEY ISSUES
Nausea, Vomiting,
Appetite Loss and Excess Stomach Acid
Maintaining Hydration
The Importance of
Phosphorus Control
All About
Hypertension
All About
Anaemia
All About Constipation
Potassium Imbalances
Metabolic Acidosis
Kidney Stones
SUPPORT
Coping with CKD
Tanya's Support Group
Success Stories
SYMPTOMS
Alphabetical List of Symptoms and Treatments
Fluid
and Urinary Imbalances (Dehydration, Overhydration and Urinary
Issues)
Waste Product Regulation Imbalances (Vomiting, Appetite Loss, Excess
Stomach Acid, Gastro-intestinal Problems, Mouth Ulcers Etc.)
Phosphorus and Calcium Imbalances
Miscellaneous Symptoms
(Pain, Hiding Etc.)
DIAGNOSIS:
WHAT DO ALL THE TEST RESULTS MEAN?
Blood Chemistry: Kidney Function, Potassium, Other Tests
(ALT, Amylase, (Cholesterol, Etc.)
Complete Blood Count (CBC):
Red and White Blood Cells: Anaemia and Infection
Urinalysis (Urine Tests)
Other Tests: Ultrasound, Biopsy, X-rays etc.
Renomegaly (Enlarged Kidneys)
Which
Tests to Have and Frequency of Testing
Factors that Affect Test Results
Normal Ranges
International and US Measuring Systems
TREATMENTS
Which Treatments are Essential
Finding a Good Vet and Record Keeping
Fluid and Urinary Issues (Fluid Retention, Infections, Incontinence,
Proteinuria)
Waste Product Regulation
(Mouth Ulcers, GI Bleeding,
Antioxidants,
Adsorbents, Azodyl, Astro's CRF Oil)
Phosphorus, Calcium and PTH (Calcitriol)
Miscellaneous Treatments: Stem Cell
Transplants, ACE Inhibitors - Fortekor, Steroids, Kidney Transplants)
Antibiotics and Painkillers
Holistic Treatments (Including Slippery Elm Bark)
ESAs (Aranesp, Epogen etc.) for Severe Anaemia
General Health Issues in a CKD Cat: Fleas, Arthritis, Dementia,
Vaccinations
Tips on
Medicating Your Cat
Obtaining Supplies Cheaply in the UK, USA and Canada
Working with Your Vet
DIET & NUTRITION
Nutritional Requirements of CKD Cats
The B Vitamins (Including
Methylcobalamin)
What to Feed (and What to Avoid)
Persuading Your Cat to Eat
Food Data Tables
USA
Canned Food Data
USA
Dry Food Data
USA
Cat Food Manufacturers
UK
Canned Food Data
UK
Dry Food Data
UK Cat Food Manufacturers
2007 Food Recall USA
FLUID THERAPY
Intravenous Fluids
Subcutaneous Fluids
Tips on Giving
Subcutaneous Fluids
How
to Give Subcutaneous Fluids with a Giving Set
How
to Give Subcutaneous Fluids with a Syringe
Subcutaneous Fluids - Winning Your Vet's Support
Dialysis
RELATED DISEASES
Heart Problems
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Diabetes
Polycystic Kidney Disease (PKD)
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OBTAINING SUPPLIES CHEAPLY
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SAYING GOODBYE
Saying Goodbye
The
Final Hours
Coping with Your Loss
Other People's Losses
MISCELLANEOUS
Prevention
Research
Canine Renal
Failure
Other Illnesses (Cancer, Liver) and
Behavioural Problems
Diese Webseite auf Deutsch
SITEOWNER (HELEN)
My
Three CKD Cats: Tanya, Thomas and Ollie
My Multi Ailment Cat,
Harpsie
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Home >
Siteowner
> My Three CKD Cats:
Tanya, Thomas and Ollie
Overview
-
Tanya, Thomas and Ollie were three of our cats, who all
developed CKD.
-
Although they did all eventually die of the disease,
their experiences and quality of life throughout the illness were very
different.
Tanya's Story
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Tanya was a Blue Persian who came to live with us at
the age of ten weeks. She was a happy cat who enjoyed good health,
although she was rather small (she averaged around 6lbs for most of her
life) and quite timid. Her only real health problem was the occasional tooth abscess which
required removal of the offending tooth - she had three of these over the
years. In
September 1998 at the age of 12, Tanya went for her annual vaccinations and
check-up. Earlier that year her coat, previously beautiful and glossy, had
become dry and was showing signs of dandruff which the vet had ascribed to
her advancing years. During this visit the vet thought she might have the first signs of CKD but
said there was nothing that could be done at that stage - she
could manage another five weeks or five years. We were very upset but
Tanya seemed stable and we naturally were hoping that she would manage
five years. At
the end of October 1998 Tanya and I returned to the surgery because she had
lost more weight and her spine was very prominent and bony. This time we
saw a different vet and blood tests were run and the vet told me Tanya had CKD. She felt Tanya had
less than a year to live - I was heartbroken. We were given steroids and
antibiotics and prescription food as treatment. Tanya went downhill fast. She
soon lost her appetite and lost a lot of weight. She had very bad mouth
ulcers and muscle weakness; there were also signs of a heart murmur. We
syringe fed her on particularly bad days. She was obsessed with drinking
water, preferably from a sink. Tanya rallied and
made it through Christmas 1998 (when she enjoyed her Christmas turkey) and
the New Year; by this time she was receiving steroid injections from the
vet every other day. On 10 January 1999 we realised Tanya was suddenly
unable to urinate; her kidneys had completely seized up. The vet kindly
came to our home and put Tanya to sleep. Tanya was buried in the garden
she loved so much; she only weighed 3lbs when she died, having lost half
of her bodyweight. We were completely
heartbroken.
Thomas's Story
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Thomas,
a black DSH, was the local stray, who was already in the neighbourhood
when we arrived in 1994. Tanya would not allow Thomas in her garden and he
used to run away from us if we tried to get near him, but our next-door
neighbour used to feed him every day. She told us he was feral and that
she could not get near him either. In
the spring of 1999 our neighbour told us she was moving into sheltered
housing and could not take Thomas with her. We decided to try and trap Thomas and tame him if possible
so that he could have a proper home. It took more than
a week (he was very wily!) and during the attempts I noticed that Thomas
had an overpowering aroma. We caught Thomas in
June 1999 and took him to the vet where they discovered that the smell was
coming from severely abscessed teeth; he had three removed. Apart from
that, he was basically healthy - his kidney values were very slightly
elevated but my vet ascribed that to the stress of being trapped and his
dental problems, and they
reverted to normal later on. We soon discovered
that Thomas was not feral, he loved living with us and settled in well. In
November, however, he had an accident which required surgery, and during
the pre-surgery blood work my vet discovered that Thomas's kidney values
were sky high. She recommended a special prescription diet and regular steroid
injections every three-four weeks. Thomas had his
first steroid jab in early December. He refused point blank to eat the
prescription food but he acted perfectly normally, showing no signs of
illness whatsoever, until the weekend of 18/19 December. On
the Saturday, Thomas went off his food a little, though he did eat some
fish in the evening. On the Sunday he refused to eat and by the evening he
was also refusing to drink; he sat hunched up and his breath had a
distinctive and unpleasant aroma. Basically, he had
crashed,
so we should have taken him in over the weekend as an emergency, but
I didn't know then that a crash was an emergency. We went to
the vet as soon as the clinic opened on Monday. Thomas was severely dehydrated and
my vet put him on IV fluids. She tested his blood and the numbers were
incredibly high (his urea was 86 - US: BUN 241); he was also severely
anaemic (his PCV was 18%, but since he was dehydrated, it was probably
much lower than that in reality). She felt he would die if he
couldn't be persuaded to eat; and even if he ate it would be touch and go
- she feared that once he was off IV he would crash again.
We were not online when Tanya was ill but by this time we were, so I went
home and searched on the internet and found some online support. The
people there recommended sub-cutaneous fluids once Thomas was home from
the vet's. I spoke to my vet who was
initially rather sceptical about the idea but eventually she agreed to
teach me how to do them for home use. Thomas had IV
treatment for four solid days and nights, yet sadly his numbers did not
fall; his urea was still 86 (US: BUN 241). However, he had at least begun
to eat a little, so he came home on Christmas
Eve, and he ate well and seemed reasonably healthy. Unfortunately I got
the 'flu over the New Year and was unable to give him any fluids, with the result that he
crashed again and was put on IV once more on 6 January, this time for
three days. After Thomas came home, his anaemia was
really severe and he was very weak, and he would only eat ham. We persevered
with some of the treatments we learnt about; and by mid February he was doing
really well. His blood work results
improved, so he was now
considered to be a "medium numbers kitty" (his creatinine level
was 3.47). He ate well,
groomed himself, went out for walks and to look at him you would not have known
he was terminally ill.
Thanks to Eprex, his anaemia was completely under control. Update
August 2000 It
is with great sadness that I write that we lost Thomas on 11 August 2000.
His blood work at the end of July showed a dramatic worsening, which came
as a shock to us and our vets, because he was acting so well. He suddenly
crashed on 10 August and we made the painful decision to let him go on 11
August, when it was apparent that this time he could not fight back; like
Tanya, he was unable to urinate. The
vet came to our home and helped him cross - it was very peaceful. He had
been acting like a healthy cat right up until 10 August, even going out
for a long walk on 9 August and returning to eat a large dinner (Thomas's
appetite was always pretty good apart from when his anaemia was severe).
Although it was a shock for us, I am glad for Thomas's sake that he was
able to enjoy life right up until the end and that his last day was spent
looking out at the garden he loved.
Although we miss Thomas terribly, there is
no guilt. We know we did our best for him, and he and we greatly enjoyed
2000, the year we thought he would not live to see. If I had known he
would leave this year (it seemed so unlikely when I was creating the
site), I would not have named this site solely in Tanya's honour - Thomas
taught me so much about life and love, and about strength and not giving
up. My vet said he was the bravest cat she had ever known. Thomas also
enabled me to learn all I know about CKD, and this site is as much a
tribute to him as it is to Tanya. One
day, when I feel able, I will create a memorial page for Thomas.
Right now it is just too painful.
Here's Thomas
in May 2000 (he does have ears really, I just accidentally cut them off!)
These pictures of Thomas below were taken in early August
2000, approximately one week before he died. I think they show how well a
cat receiving treatments can look and feel, even towards the end.
Ollie's Story
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Ollie was a cream Persian, like our first male cat,
Harpsie. A
family member was so smitten with Harpsie that he went to the shelter to
adopt a cream Persian and ended up with two, Ollie and his mother, Cleo.
Ollie was treated successfully for mammary cancer in 2006. He had a bad
time of it in 2007. First, his mother, Cleo, died of cancer. Then his
human moved to be with his fiancée, so Ollie had to get used to being an
only cat for the first time in his life, move to a new home and get used
to three new humans, including two children. The children loved Ollie, but
he had never lived with children before. And the fiancée didn't like Ollie
much.
In January 2008, Ollie's right eye had to be removed. He had suffered from
herpes for many years, and the eye just erupted. Ollie was fifteen years
old and very anaemic, but he made it through the surgery, and adapted to
living with one eye.
Unfortunately, in March 2008 Ollie's human split up with his fiancée. He
had nowhere to live, and nor did Ollie. We offered to take Ollie in when
we returned from the USA. In the meantime, Ollie went to live for three
months at the cattery we use when we go on holiday.
When
we returned from the USA, Ollie came to live with us, in late June 2008,
one week before his sixteenth birthday. The first time I saw him (left), he
was all blond and cute and fluffy. And then I picked him up. He was so
thin that I could put my (small) hands around his waist and they
overlapped! I could feel his spine, yet he ate like a horse. He was also
urinating and drinking a lot. I knew immediately that he must have a
medical problem. I booked an appointment with my vet for the next day.
That evening it became apparent that Ollie had additional problems in the
form of being very wobbly, vomiting, diarrhoea and pooping outside the
litter box. The latter was the main reason the fiancée hadn't liked
Ollie. Ollie was installed in our guest bedroom with puppy pads and
Catpaper all over the bed and carpet for easy clean ups. He seemed like a
very laidback kind of little guy, but then blond Persian boys do seem to
be like that.
Tests at the vet's the next day showed that Ollie weighed 5.3 lbs when in
the vet's opinion he should have weighed at least 8.5, and 9 lbs would
probably be his ideal weight. He had a heart murmur at level 4. He also
had CKD. His BUN was 80, his creatinine 2.81, his phosphorus 8.04 and his
potassium 3.5. He was also anaemic, with PCV at 23%. Tests for an
overactive thyroid were sent to the lab.
Ollie's
biggest obvious problem was his weak back legs. He couldn't walk properly,
he twitched a lot, and while the vet was on the phone to me, poor Ollie fell off
the window ledge. I asked the vet if we could start him on a potassium
supplement. She didn't think it was necessary because his level was
exactly at the bottom of the range for the lab she used but she agreed to
let me give him one. After two days of the potassium supplement, Ollie could walk
normally again! Ollie was also given a phosphorus binder, B vitamins and
iron and a prescription diet which he actually seemed to enjoy.
Surprisingly, Ollie's T4 (thyroid function test) came back as normal. Yet
he had so many signs of hyperthyroidism that I found this hard to believe.
So we did a Free T4 test, which showed that in fact, Ollie was indeed
hyperthyroid. We started him on medication for that too.
After
five weeks of treatment Ollie had largely stopped vomiting and having
diarrhoea and had gained 0.8 lbs. He was eating well and seemed happy - he
was a very placid, sweet-natured, cute little guy who loved his cuddles.
He got on pretty well with his new sisters, Indie and Karma.
Ollie continued to gain weight and enjoyed the summer. Eventually he got
up to 6.8 lbs, a great improvement on the 5.3 lbs he had weighed when he
first came to live with us. He developed high blood pressure but we were
able to control this with medication.
Unfortunately we struggled to control Ollie's anaemia. His HCT would go up
a little, then fall back down again. We discovered that his HCT had
actually been extremely low, only 18%, in October 2007, but his vets at
that time hadn't even mentioned it to his human! So his anaemia was
chronic, yet we struggled to find the cause. My vet suspected cancer, but
Ollie was still too frail for invasive tests.
Towards the end of October 2008 Ollie went to the vet to check his blood
pressure and anaemia. His blood pressure was normal, but his anaemia had
worsened once again, from 23% to 18%. I asked the vet to trim Ollie's
claws since they were catching on things. Ollie collapsed! He had to be
placed in the oxygen tent to recover. The vet said he simply couldn't pump
enough oxygen around his body to cope with the stress he was under
because of the anaemia and heart issues. She said he was balanced on a knife edge.
Ollie
came home and seemed to be none the worse for his collapse. The photo to
the right was taken on 4 November 2008 when he seemed fine. A couple of
days later, however, he seemed subdued, was grinding his teeth and was not
eating much, so the following day we went to the vet's. Ollie's HCT was
back up to 24% which was encouraging. However, we found blood in his urine
so he had to go on an antibiotic.
Ollie seemed to improve somewhat on the antibiotic, though there continued
to be obvious blood in his urine. I was concerned about him and made an
appointment with the vet for 13 November. Sadly, on the evening of 12
November, Ollie suddenly collapsed. He was struggling to breathe and spent
the night in the oxygen tent. Tests on 13 November showed that Ollie's
poor little body was shutting down. He was in heart failure and his kidney
values had suddenly worsened dramatically, whilst his HCT continued to
fall throughout the day. His blood pressure kept falling. The vet said we
could try to help if it was his heart or his kidneys, but trying to treat
both conditions when Ollie was so frail would simply not work. The vet
helped Ollie to cross that evening, with us and his original human
present. He was gone within five seconds, so he clearly had given his all
and had no reserves left.
I still feel a bit cheated with Ollie. If only his original vets had
treated his anaemia and other health issues back in 2007. If only he had
never got so thin with the undiagnosed hyperactive thyroid, which needed
additional tests for accurate diagnosis. If only we had returned earlier
from the USA
so he could have come to live with us sooner. But we did the best we
could in the circumstances, and I think he felt safe and happy and loved
with us. I miss my cute little guy.
*****
"'Cause I
miss you, body and soul, so strong
That it takes
my breath away.
And I breathe
you into my heart
And pray for
the strength to stand today.
'Cause I love
you, whether it's wrong or right,
And though I
can't be with you tonight,
You know my
heart is by your side."
Daniel
Bedingfield
Also in loving
memory of Alex

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This page last updated: 14 July 2010
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